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6월 10일 (토)

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https://us06web.zoom.us/j/86716953250

Cellular healing of ancestral trauma and interdimensional blockages DNA activation

Learn how to integrate and break patterns of suffering pain and contraction

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Cellular healing of ancestral trauma and interdimensional blockages  DNA activation
Cellular healing of ancestral trauma and interdimensional blockages  DNA activation

시간 및 장소

2023년 6월 10일 오전 11:00 – 오후 1:05 GMT-7

https://us06web.zoom.us/j/86716953250

이벤트 소개

Our life experiences may be passed on to our children and our  children’s children through our genes. The open question is, can we  make a difference? Will healing our own traumas prevent them being  delivered at the baby carriage of our grandchildren? Scientists are now  revealing that they have discovered how this genetic inheritance can be  turned on or off. The good news: healing personal trauma will make a  huge difference to future generations.

According to epigenetics—the study of inheritable changes in gene  expression not directly coded in our DNA—our life experiences may be  passed on to our children and our children’s children. Studies on  survivors of traumatic events have suggested that exposure to stress may  indeed have lasting effects on subsequent generations. But how exactly  are these genetic “memories” passed on?

Lecture

How trauam is created 

level of somatic impact of trauma 

the body on trauma 

how information is imput into DNA 

How Trauma is passed down through generations 

How trauma affects our epigentic intake and expression. 

The Beauty and geometry of DNA

chrystalization process of Frequency

Alchemy and Genetic Mutations

Workshop

Identifying patterns ancestral Patterns

clearning ancestral patters

The inner child speaks for them all

how healing functions

Intuition and Expansion 

The Morphogentic field 

Alchemy process

we will do an in depth review of programs related to the hologram overlay 

how it ffects the phyche of humanity and how to break these paterns in order to embody an epansive field 

or cocreaton cross dimensionaly. 

this event will be recorded and available for unlimited replays. 

Unconditional Love

 Gerldine 

A new Tel Aviv University study pinpoints the precise mechanism that  turns the inheritance of environmental influences “on” and “off.” The  research, published last week in Cell and led by Dr. Oded  Rechavi and his group from TAU’s Faculty of Life Sciences and Sagol  School of Neuroscience, reveals the rules that dictate which epigenetic  responses will be inherited, and for how long.

“Until now, it has been assumed that a passive dilution or decay  governs the inheritance of epigenetic responses,” Dr. Rechavi said. “But  we showed that there is an active process that regulates epigenetic  inheritance down through generations.”

Passing stress from one generation to the next

Researchers have been preoccupied with how the effects of stress,  trauma, and other environmental exposures are passed from one generation  to the next for years. Small RNA molecules—short sequences of RNA that  regulate the expression of genes—are among the key factors involved in  mediating this kind of inheritance. Dr. Rechavi and his team had  previously identified a “small RNA inheritance” mechanism through which  RNA molecules produced a response to the needs of specific cells and how  they were regulated between generations.

“We previously showed that worms inherited small RNAs following the  starvation and viral infections of their parents. These small RNAs  helped prepare their offspring for similar hardships,” Dr. Rechavi said.  “We also identified a mechanism that amplified heritable small RNAs  across generations, so the response was not diluted. We found that  enzymes called RdRPs are required for re-creating new small RNAs to keep  the response going in subsequent generations.”

Most inheritable epigenetic responses in C.elegans worms were found  to persist for only a few generations. This created the assumption that  epigenetic effects simply “petered out” over time, through a process of  dilution or decay.

“But this assumption ignored the possibility that this process  doesn’t simply die out but is regulated instead,” said Dr. Rechavi, who  in this study treated C.elegans worms with small RNAs that target the  GFP (green fluorescent protein), a reporter gene commonly used in  experiments. “By following heritable small RNAs that regulated GFP—that  ‘silenced’ its expression—we revealed an active, tuneable inheritance  mechanism that can be turned ‘on’ or ‘off.’”

The scientists discovered that specific genes, which they named  “MOTEK” (Modified Transgenerational Epigenetic Kinetics), were involved  in turning on and off epigenetic transmissions.

“We discovered how to manipulate the transgenerational duration of  epigenetic inheritance in worms by switching ‘on’ and ‘off’ the small  RNAs that worms use to regulate genes,” said Dr. Rechavi. “These  switches are controlled by a feedback interaction between  gene-regulating small RNAs, which are inheritable, and the MOTEK genes  that are required to produce and transmit these small RNAs across  generations.

“The feedback determines whether epigenetic memory will continue to  the progeny or not, and how long each epigenetic response will last.”

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